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Ostarine is less suppressive than Anavar, outperforms it in an anabolic capacity, and displays a significantly lower incidence of side effects and androgenic activity in the body. In comparison to Anavar, Ostarine has no measurable bioavailability and appears to exert no androgenic activity in vivo due to the lack of a testosterone conjugate in the urine. Furthermore, there is no evidence to indicate that Ostarine has any effect on serum concentrations of testosterone and free estradiol.Ostarine is generally metabolized by the liver via CYP1A2 through mono- and dihydro-Ostarine hydroxylation, resulting in the formation of 3,10-dihydroxy-4,6-dienyl-6,7,10-dihydroxy-6,7,10-dienoic acid and (O)-6,7,10-dihydro-(O)-6,7,10-dihydro-6,7,10-dienoic acid. Dihydro-6,7,10-dihydroxy-6,7,10-dienoic acid forms a hydroxymethylated derivative with more potent androgenic activity. This hydroxy product is known as 4,10 -octenyl-6,7-dienoic acid. The formation of this molecule from Ostarine provides an avenue through which to obtain 4α-Dihydro-6,7,10-dihydro-6,7,10-dienoic acid for the metabolism of DHEA. In the absence of androgens, there is no direct conversion of these products. In men, these products form either 4 or 7 hydroxy groups via the formation of 4α-Dihydro-6,7,10-dihydro-6,7,10-dienoic acid with different conjugate structures. These products are present in the urine and the blood, but can be eliminated by deamination and/or by conjugating with a deuterium (10-16N) salt. In general, the lower concentration of 4,10-dihydro-6,7,10-dienoic acid in the blood indicates a higher bioavailability of these products. The conjugates are used clinically for the treatment of hypogonadism and/or hyperandrogenism.Ostarine may be metabolized by the liver to 4,10-dihydroxy-6,7,10-dienoic acid and 3Related Article: